How To Use Cardiac output In A Sentence
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This is more likely following infarction when cardiac output may be reduced and therefore tissue perfusion impaired.
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If cardiac output is compromised, hypoxia of the peripheral tissues may cause lactic acidosis to develop or worsen.
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Heart rate, cardiac output, and minute ventilation increase under hyperthermic conditions, while visceral perfusion decreases.
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Later that day, he developed cardiogenic shock, requiring numerous pressor agents and the aortic balloon pump to maintain his cardiac output.
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Objective To determine the normal value of cardiac output measured by reconstructed impedance cardiogram.
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Be cautious in prescribing diuretics, as excessive diuresis may reduce stroke volume and cardiac output.
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Nitroglycerin reduces BP by reducing preload and cardiac output, undesirable effects in patients with compromised cerebral and renal perfusion.
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Pathogenesis of Hypertension Blood pressure is dependent upon two factors: cardiac output and peripheral resistance.
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With the fall intravascular volume, there is diminished venous return to the right heart, low cardiac output, and a drop in systemic blood pressure.
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Beta-blockers (nadolol, atenolol, metoprolol, propranolol, and others) reduce cardiac output (negative inotropic and chronotropic effects) but increase peripheral resistance.
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Patients at risk may include those with a history of atherosclerosis, multiple cardiovascular risk factors, advanced age, impaired cardiac output, and/or known or suspected hyperviscosity, hypercoagulable disorders, and prolonged periods of immobilization.
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It may also become possible to boost oxygen uptake, cardiac output and metabolic efficiency.
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This keeps patients close to waking, even when extractions or other invasive procedures are needed, thus maximizing cardiac output and tissue perfusion and maintaining blood pressure.
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In congestive heart failure, it is diminished because of low cardiac output and reduced arterial distending pressure.
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Miller reported that low cardiac output syndrome and atrial dysrhythmias were associated with prolonged stays in the ICU.
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The principal factors inducing right ventricular strain, hypertrophy, and failure can be stated very simply: (1) pulmonary hypertension, from one cause or another; and (2) secondary influences throwing a burden on the right heart, such as anoxia, increased blood volume, polycythemia, increased cardiac output, disordered breathing mechanics.
Dickinson W. Richards - Nobel Lecture
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Pathogenesis of Hypertension Blood pressure is dependent upon two factors: cardiac output and peripheral resistance.
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Ischaemia secondary to reduced cardiac output may contribute to the risk of visceral damage.
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Fetal echocardiography revealed that a large portion of the fetal cardiac output perfused the Sacrococcygeal Teratoma.
Sacrococcygeal Teratoma (SCT), Sacrococcygeal Tumor Treatment
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The pump was stopped smoothly open heart surgery finished. No postoperative low cardiac output syndrome happened.
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It may also become possible to boost oxygen uptake, cardiac output and metabolic efficiency.
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a resting adult has a cardiac output of about three quarts a minute
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Pathogenesis of Hypertension Blood pressure is dependent upon two factors: cardiac output and peripheral resistance.
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High output failure occurs when the cardiac output of the heart remains significantly elevated for a long period.
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An infusion of IV normal saline frequently corrects hypotension and increases cardiac output.
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Microvascular vasoconstriction is the underlying process and is precipitated by splanchnic hypoperfusion due to depressed cardiac output or renal or hepatic disease.
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The decrease in cardiac output that occurs with heart failure is associated with a decrease in blood pressure that activates baroreceptors located in the aortic arch and carotid arteries.
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Factors contributing to the additional improvement include increases in oxygenation, right ventricular function, cardiac output, number of sarcomeres, and muscle mass, and a decrease in glucocorticoid use.
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The study was inspired by ongoing research at Penn into the physical forces of cells and the well-known fibrotic rigidification and impairment of cardiac output that follows a heart attack or other trauma to the heart muscle.
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Partial CO_2 rebreathing is a novel method for cardiac output measurement.
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The decrease in cardiac output that accompanies heart failure also leads to significant reductions in renal blood flow.
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This is more likely following infarction when cardiac output may be reduced and therefore tissue perfusion impaired.
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arterial pressure is the product of cardiac output and vascular resistance
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Many forms and degrees of failure were defined, and their responses to treatment measured: limited or fixed cardiac output, not responding to exercise; left ventricular failure with pulmonary hypertension; right ventricular failure with systemic venous hypertension; the congestive state with high blood flow, and with low; very low cardiac output without congestion; the dynamic effects of cardiac arrhythmias; the circulation in constrictive pericarditis31, 32.
Dickinson W. Richards - Nobel Lecture
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In these patients, the heart rate, cardiac output, and blood pressure rise appropriately in response to exercise.
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Patients at risk may include those with a history of atherosclerosis, multiple cardiovascular risk factors, advanced age, impaired cardiac output, and / or known or suspected hyperviscosity, hypercoagulable disorders, and prolonged periods of immobilization.
Undefined
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There was no cardiac output and resuscitation was abandoned at 1.55 a.m.
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These mice are characterized by polycythemia but have normal blood pressure, heart rate, and cardiac output.
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PAOP 14-18mm Hg. This was achieved by a combination of bolus fluid administration, use of frusemide or dobutamine, depending on the perfusion, urine output, measured filling pressures and cardiac output (if
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Thus, large doses of the drug may be required in some patients with septic shock, possibly due to α-receptor down-regulation in sepsis. 111 Cardiovascular Effects Norepinephrine therapy usually causes a clinically significant increase in MAP attributable to its vasoconstrictive effects, with little change in HR or cardiac output, leading to increased systemic vascular resistance.
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